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Decision-Making Algorithm in Otorhinolaryngology & Head and Neck Surgery

Chronic Cough

The cough that outlasts eight weeks — a duration-first, treatable-traits sweep from chest X-ray and the big three to cough hypersensitivity and the neuromodulator-and-speech-therapy endgame

Step 1

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Presentation & Duration

Classify by duration — acute <3 wk · subacute 3–8 wk · chronic >8 wk (this chapter)

The first and most decisive question in any cough is how long it has lasted, because duration, more than any other feature, sorts the differential. Acute cough (under 3 weeks) is dominated by viral upper-respiratory infection, acute bronchitis, influenza, COVID-19, and pneumonia — self-limiting or infective, and managed accordingly. Subacute cough (3–8 weeks) is most often post-infectious, the lingering cough after a resolved infection, but also captures pertussis, a first presentation of asthma, sinusitis, and bronchitis. Chronic cough (over 8 weeks) is the subject of this chapter and a different problem altogether.

Crossing the eight-week threshold reframes the clinical task: the question is no longer "which infection" but "which treatable trait, or which cough hypersensitivity, is driving a cough that should have stopped and has not". The duration cut-off is not arbitrary — it marks the point at which post-infectious and self-limiting causes have largely declared themselves, and a systematic, multidisciplinary evaluation becomes worthwhile. Establishing that a cough is genuinely chronic is therefore the gateway to the entire algorithm, and it is worth pinning down the true onset and time-course before launching the workup.

  • Acute <3 wk — viral URI, acute bronchitis, influenza, COVID-19, pneumonia (mostly self-limiting/infective)
  • Subacute 3–8 wk — post-infectious cough, pertussis, asthma, sinusitis, bronchitis
  • Chronic >8 wk — the full treatable-traits / cough-hypersensitivity evaluation
  • Duration reframes the differential away from infection toward treatable traits

Key statistics

  • Chronic = over 8 weeks

    >8 weeks defines chronic cough

    The 8-week threshold is the gateway to the chronic-cough algorithm — it marks where self-limiting and post-infectious causes have largely declared themselves.

    PMID 31515408

Cough by duration

Duration is the first and most decisive sort

  1. Acute

    < 3 weeks — viral URI, acute bronchitis, influenza, COVID-19, pneumonia

    mostly self-limiting / infective

  2. Subacute

    3–8 weeks — post-infectious cough, pertussis, asthma, sinusitis

  3. Chronic

    > 8 weeks — treatable traits + cough hypersensitivity

    the full evaluation in this chapter

★ High-yield pearls (chapter-wide)

  • Chronic cough is defined by duration — over 8 weeks — and that single number reframes the differential away from acute infection toward the treatable traits and cough hypersensitivity.
  • A chest radiograph is mandatory in every chronic cough, and an ACE-inhibitor must be stopped before anything else — the two cheapest, highest-yield steps in the whole algorithm.
  • In ENT practice, flexible nasolaryngoscopy is done early — it screens for malignancy, vocal-fold pathology, paradoxical fold motion, and the laryngeal signs of reflux and sensory neuropathy in one look.
  • Upper-airway cough syndrome, reflux, asthma and non-asthmatic eosinophilic bronchitis, and cough hypersensitivity account for the great majority of chronic cough with a normal chest X-ray.
  • Most chronic cough is multifactorial — finding one treatable trait does not excuse you from looking for the others, and incomplete response usually means a second contributor is still active.
  • Non-asthmatic eosinophilic bronchitis coughs without wheeze, has normal spirometry and a normal airway-responsiveness test, and is caught only by sputum eosinophils or FeNO — yet it responds beautifully to inhaled steroids.
  • Cough hypersensitivity syndrome is the modern unifying concept — laryngeal hypersensitivity and central sensitisation, often post-viral, with the patient who says 'everything triggers my cough': talking, laughing, eating, perfume, cold air.
  • Neurogenic cough is a diagnosis of exclusion that responds to neuromodulators — gabapentin, pregabalin, or amitriptyline — not to more acid suppression or more antihistamine.
  • Speech-language-pathology cough-suppression therapy is a core treatment, not an afterthought — it durably reduces refractory cough and is paired with, not replaced by, neuromodulators.
  • Refractory chronic cough (a treatable trait found and treated, but cough persists) and unexplained chronic cough (no cause found) are distinct entities — and both are the natural home of P2X3 antagonists such as gefapixant.

Evidence base

2 sources
  1. HIGH

    Morice AH, Millqvist E, Smith JA · Eur Respir J · 2020Clinical practice guidelinePMID 31515408

    The European Respiratory Society guideline defining chronic cough (>8 weeks), the diagnostic evaluation, the treatable traits, cough hypersensitivity, and the role of neuromodulators and speech therapy.

  2. HIGH

    Pratter MR · Chest · 2006Clinical practice guidelinePMID 16428693

    ACCP overview establishing the anatomic-diagnostic protocol and the common treatable causes of chronic cough — upper-airway cough syndrome, asthma, non-asthmatic eosinophilic bronchitis, and reflux.

Decision tree

Duration sets the frame; the red-flag screen, a mandatory chest X-ray, and the wet-versus-dry fork structure the workup. The treatable traits and the hypersensitivity phenotype route the dry cough to one of ten pathways.

Chronic cough (>8 weeks)Red-flag screen?No red flagRed flagChest X-ray → productive or dry?Normal filmTreatable trait / phenotype?Red flag (A)Haemoptysis, weight loss, neck mass, abnormal chest —urgent imaging & referralACE-I (B)Stop & substitute the ACE inhibitor; reassess at 4–12 weeks— often curativeProductive (C)Wet cough → TB, bronchiectasis, COPD; sputum, GeneXpert,CT, lung functionUACS (D)Postnasal drip → empiric saline + intranasal steroid +antihistamine (treat-to-confirm)Reflux (E)GERD/LPR → diet, weight, alginate; selective PPI;impedance-pH if refractoryEosinophilic (F)Asthma / NAEB → FeNO & sputum eosinophils; inhaledcorticosteroidsOther traits (G)Irritant, rheumatologic, cardiac, structural-airway, OSA —sweep before 'hypersensitive'ILO (H)Paradoxical fold motion → laryngoscopy + provocation;respiratory retraining, not inhalersHypersensitivity (I)'Everything triggers my cough' → neuromodulators + speechtherapyRefractory (J)SLP cough suppression + neuromodulators + gefapixant;6–12-wk MDT loopPathways:A Red flagB ACE-IC ProductiveD UACSE RefluxF EosinophilicG Other traitsH ILOI HypersensitivityJ Refractory
Step 1

Disclaimer

For educational purposes only. Not for clinical use. This platform is an instructional resource intended to support learning about clinical decision-making and the interpretation of investigations. Clinicians remain completely responsible for the interpretation of findings, the formulation of a differential diagnosis, and any clinical decision. Nothing in this application replaces individualized assessment, hands-on training, expert consultation, or established practice guidelines.

Not for profit effort by

Dr. Prahlada N.B

  • MBBS (JJMMC), MS (PGIMER, Chandigarh)
  • MBA in Hospital & Healthcare Management (BITS, Pilani)
  • Postgraduate Certificate in Technology Leadership and Innovation (MIT, USA)
  • Executive Programme in Strategic Management (IIM, Lucknow)
  • Senior Management Programme in Healthcare Management (IIM, Kozhikode)
  • Advanced Certificate in AI for Digital Health and Imaging Program (IISc, Bengaluru)

Supporting organisations

  • Karnataka ENT Hospital and Research Centre (R)
  • Champions Educational and Medical Society (R)
  • Amogh Foundation